Honeysuckle Paj Extract 25% Chlorogenic Acid | 84603-62-3

Honeysuckle extract yog muab rho tawm los ntawm honeysuckle, tseem hu ua Japanese honeysuckle lossis honeysuckle. Nws yog lub npe hu ua cov tshuaj tua kab mob zoo tshaj plaws thiab txo qhov mob. Nws yog ib qho ntawm cov tshuaj suav tshuaj ntsuab paub ntau dua.

Lub Compendium ntawm Materia Medica hu ua honeysuckle tsuas yog vim nws cov paj pib dawb (nyiaj) thiab tom qab ntawd tig daj (kub) thaum lub paj tawg tag. Vim nws cov khoom siv tshuaj tshwj xeeb thiab ntau cov txiaj ntsig, nws yog siv tsis yog tsuas yog siv tshuaj xwb tab sis kuj hloov tshuaj yej vim nws iab-qab zib saj thiab aroma.

Tsis tas li ntawd, kev noj haus tsis tu ncua yuav tsis ua mob plab, nws tuaj yeem txo qhov mob sai sai los ntawm kev tshem tawm cov dej noo thiab co toxins, thiab honeysuckle muaj chlorogenic acid, uas tuaj yeem txo qhov kev pheej hmoo ntawm gallstones.

Kev ua tau zoo thiab lub luag haujlwm ntawm Honeysuckle Paj Extract 25% Chlorogenic Acid: 

Kev tiv thaiv kab mob plawv

CGA (chlorogenic acid, CGA) raws li cov dawb radical scavenger thiab antioxidant tau raug pov thawj los ntawm ntau qhov kev sim ll J. Qhov kev ua haujlwm lom neeg ntawm CGA tuaj yeem muaj kev tiv thaiv ntawm cov hlab plawv.

Anti-mutagenic thiab anti-cancer teebmeem

Tsiaj thwmsim qhia tau hais tias CGA muaj kev tiv thaiv thiab inhibitory cuam tshuam rau qhov tshwm sim ntawm gastric mob cancer thiab txoj hnyuv.

Cov tshuaj tiv thaiv mutagenic thiab tiv thaiv qog noj ntshav ntawm CGA tuaj yeem cuam tshuam nrog cov hauv qab no: Pro-oxidation: Jiang li al. pom tias CGA yog ib qho pro-oxidant nyob rau hauv ib puag ncig alkaline, uas tuaj yeem ua rau cov qog hlwb los tsim cov DNA loj dua thiab ua rau muaj kev sib txuas ntawm nuclear. Cov nyhuv no yuav cuam tshuam nrog hydrogen peroxide.

Lipid-txo cov nyhuv

Intravenous tswj ntawm CGA txo qis plasma roj cholesterol thiab triglyceride ntau ntau hauv cov nas, nrog rau cov qib siab triglycerides.

Anti-leukemia nyhuv

Kev tshawb fawb hauv vitro los ntawm Chiang et al pom tias CGA tsis muaj zog tiv thaiv kab mob leukemia J. Bandyopadhyay thiab lwm yam kev tshawb fawb tau pom tias CGA tuaj yeem cuam tshuam Ber-Abl thiab c-Abl tyrosine kinase, thiab induce lub apoptosis ntawm Ber-Abl zoo hlwb suav nrog Ber. -Abl zoo blast lymphocytes hauv cov neeg mob uas mob myeloid leukemia.

Immunomodulatory teebmeem

Cov kev tshawb fawb hauv vitro tau pom tias CGA tuaj yeem tsis tsuas yog txhim kho kev loj hlob ntawm T hlwb uas tshwm sim los ntawm tus kab mob khaub thuas antigens, tab sis kuj txhawb kev tsim cov 7-IFN thiab a-IFN hauv tib neeg lymphocytes thiab tib neeg peripheral ntshav leukocytes.

Cov nyhuv hypoglycemic

Cov kev tshawb fawb ntawm Andrade-Cetto A thiab Wiedenfeld H tau lees paub tias CGA muaj cov nyhuv hypoglycemic hauv cov tsiaj, thiab nws cov nyhuv hypoglycemic nyob rau hauv 3 h tsis suav nrog qhov sib txawv ntawm glyburide [31 J. Cov txheej txheem yuav cuam tshuam rau inhibition ntawm qabzib-6. - Phosphate Transferase thiab Glucose Absorption.

Lwm tus

CGA tseem tuaj yeem cuam tshuam kev tsim cov cytokines thiab chemokines los ntawm staphylococcal exotoxin, thiab inhibit qhov contraction ntawm fibroblast collagen network thiab kev ntxhov siab los ntawm hypertrophic caws pliav-derived fibroblasts (mFs).

Adrenocorticotropic hormone (ACm) nce los ntawm cov tshuaj tiv thaiv.